Blood sugar
Naltrexone, an opiate antagonist, was administered to young obese (ob/ob) and lean mice for five weeks. Animals had continuous access to food and received 10 mg/kg SC two tmes a day with equivalent volumes of saline presented to controls. The effects on body weight, and pituitary and plasma numbers of β-endorphin-like material were measured.http://www.thebloodsugarsecretreview.com/
Naltrexone-injected obese animals gained weight more slowly within the first three weeks while the putting on weight of lean animals wasn't impacted by naltrexone. Plasma degrees of β-endorphin were been shown to be significantly higher in untreated ob/ob mice which difference increased with age (4–20 weeks). With naltrexone treatment, plasma levels in +/? mice rose and exceeded those who work in ob/ob.
Saline treatment was a stress, and pituitary β-endorphins rose 4–6 fold in ob/ob in comparison with +/?. While naltrexone reduced the amount in ob/ob pituitary towards normal, no effect on β-endorphin levels in pituitary of lean mice was obtained. In vitro studies of connection between the opiate antagonists, naloxone, on insulin secretion by isolated islets provided additional evidence of resistance of lean mice to naloxone compared to ob/ob. (IRI secretion fell only in naloxone treated ob/ob islets.) These observations support the contention that way of genetic obesity is seen as a elevated endogenous opiate levels as well as an increased sensitivity to opiate antagonists such as naltrexone or naloxone.
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